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肝昏迷(或肝性脑病)通常见于有严重肝脏疾病的患者。一般分为急性和慢性两种。急性肝昏迷的病死率约90％,慢性肝昏迷发作时的即时预后虽然稍好,但也仅能苟安一时,远期预后仍差。近年来,国外对肝昏迷的发病机制及治疗的研究取得了一些进展。现择其主要方面作一扼要介绍,供参考。肝昏迷治疗的病理生理基础到目前为止,关于肝昏迷的完整的发病机制还有些问题未得到满意的解释。某些学者仍然认为肝昏迷的主要原因是血氨,尤其是动脉血氨增高,从而影响大脑的功能。不少实验研究和临床观察继续支持这一点。但是
Hepatic coma (or hepatic encephalopathy) is usually found in patients with severe liver disease. Generally divided into two kinds of acute and chronic. The mortality rate of acute hepatic coma is about 90%. Although the immediate prognosis of chronic hepatic coma is slightly better, it can only be used for a short time and the long-term prognosis is still poor. In recent years, foreign countries have made some progress on the pathogenesis and treatment of hepatic coma. Now choose its main aspects for a brief introduction for reference. Pathophysiology of Hepatic Coma Treatment So far, there have been problems with the complete pathogenesis of hepatic coma that have not been satisfactorily explained. Some scholars still believe that the main reason for hepatic coma is blood ammonia, especially arterial ammonia, which affects the functioning of the brain. Many experimental studies and clinical observations continue to support this. but