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人类分娩动因的精确机理至今仍未完全清楚。70年代初期动物实验特别是Ligginsc 应用绵羊研究,认为胎儿丘脑下部—垂体—肾上腺轴的活动,肾上腺皮质醇的增加在分娩发动上起主导作用,认为分娩发动的启动因子源于丘脑下部。在人类也得到证明,如妊娠合并无脑儿,由于无脑儿垂体缺如或不发育,ACTH 分泌少或无,致使胎儿肾上腺不发育,皮质醇不增加,以致约40%的妊娠合并无脑儿,若不合并羊水过多症者均妊娠延期。妊娠死胎基于同样原理亦妊娠
The exact mechanism of human delivery motivation is still not fully understood. In the early 1970s, animal experiments, especially the Ligginsc sheep study, suggested that fetal hypothalamus-pituitary-adrenal axis activity and adrenal cortisol increase play a leading role in childbirth initiation, suggesting that the initiating factor of labor initiation originates in the hypothalamus. In humans have also been proven, such as no cerebral anemia in children with pregnancy, due to lack of brain pituitary absence or lack of development, ACTH secretion or less, resulting in fetal adrenal development, cortisol does not increase, so that about 40% of pregnancy without brain Children, if not combined with polyhydramnios were delayed pregnancy. Pregnancy stillbirth also based on the same principle of pregnancy