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作者研究了92例急性肾衰患者,其中54例(59%)属非尿少性急性肾衰,平均每日尿量为1280±75ml。此54例中,36例在整个住院过程中尿量一直不减少;另18例开始的4～48小时时尿少,予一剂或数剂速尿(2～10mg/kg静注)后才成为非尿少性,在临床特征与肾衰严重度上,与自然的非尿少性患者间无显著差异。结果,尿少性与非尿少性患者相比,毒素诱致的急性肾衰(4例由造影剂,15例由肾毒性抗菌素所致)是两组间唯一构成统计学显著差异的原因。发生肾衰时,比较24例尿少性与31例自然的非尿少性患者,发现后者的尿钠浓度与已滤过钠的排泄分数比前者显著为低;而尿素氮在尿与血中的比值比前者显著
The authors studied 92 patients with acute renal failure, of which 54 (59%) were non-less acute renal failure with an average daily urine output of 1280 ± 75 ml. In the 54 cases, 36 cases of urine in the entire hospital process has not decreased; the other 18 cases started 4 to 48 hours less urine, to one or several doses of furosemide (2 ~ 10mg / kg intravenous) before Become non-urinary less severe in clinical features and severity of renal failure, and no significant difference between non-oliguric and natural patients. As a result, toxin-induced acute renal failure (4 with contrast media and 15 with nephrotoxic antibiotics) was the only reason for the statistically significant difference between the two groups when compared with patients with nontremia. In the case of renal failure, 24 cases of oliguric urine and 31 cases of natural non-oliguric patients were compared and found to be significantly lower urinary sodium concentration and fractionated sodium excretion than the former; and urea nitrogen in urine and blood In the ratio of the former significant