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十二指肠溃疡的复发是由于溃疡愈合后幽门弯曲菌(Campylobacter pylori,CP)感染的持续存在,Coghlan 等认为这一事实有力地证明了 CP 在溃疡发生中的作用。CP 不能移生于小肠上皮,那么胃中的 CP 感染是怎样引起十二指肠溃疡的呢?答案很可能就足在十二指肠中出现了胃上皮化生。胃上皮化生为小肠上皮对损伤的一种反应。在人和实验动物中,十二指肠球部的胃上皮化生都与胃酸增高有关,这意味着胃上皮化生是十二指肠对其内容
The recurrence of duodenal ulcer is due to the persistent presence of Campylobacter pylori (CP) infection following ulcer healing, a fact Coghlan et al. Strongly suggests the role of CP in ulceration. CP can not migrate in the small intestine epithelium, then the stomach CP infection is how to cause duodenal ulcer? The answer is likely to be enough in the duodenal gastric metaplasia. Gastric metaplasia is a response of the intestinal epithelium to injury. In human and laboratory animals, gastric metaplasia of the duodenal bulb is associated with increased gastric acidity, which means that gastric metaplasia is the component of the duodenum