论文部分内容阅读
小叶内动脉是胰腺微循环障碍中首先受累的部位,在急性胰腺炎发生发展过程中,胰腺微循环障碍既可作为始动因素,也可作为持续和加剧损害的因素。急性胰腺炎时,胰腺微循环损伤表现为胰腺微血管痉挛、通透性改变、滋养组织灌流改变和白细胞-内皮细胞相互作用等。然而,胰腺微循环损伤的影响因子是复杂的;从分子血液流变学角度阐明胰腺微循环障碍的发生机制,对于揭示急性胰腺炎的发生机理具有重要意义。
Lobular artery is the first site of pancreatic microcirculation involved in the site of the occurrence and development of acute pancreatitis, pancreatic microcirculation can be used as a starting factor, but also as sustained and exacerbated the damage factor. Acute pancreatitis, pancreatic microcirculation damage manifested as pancreatic microvascular spasm, changes in permeability, changes in nourishing tissue perfusion and leukocyte - endothelial cell interaction. However, the influencing factors of pancreatic microcirculation damage are complex; clarifying the mechanism of pancreatic microcirculation disorder from the perspective of molecular hemorheology is of great significance for revealing the pathogenesis of acute pancreatitis.