论文部分内容阅读
维生素A的积蓄和胡萝卜素合成维生素A的过程均在肝脏进行。维生素A由肝脏进入各器官和组织。有资料指出在维生素A不足和缺乏时,机体免疫指标——吞噬指数和补体滴度——显著降低。维生素A能增强机体对某些毒物和毒素的抵抗力,并有抗营养障碍的特性。它可激活胰岛素,促进糖原在肝脏的储存,而肝脏对致病因素作用的耐受性取决于肝脏糖原生成作用的程度。为探讨病毒性肝炎患者血清维生素A与胡萝卜素含量的动态改变,作者检查了14～66岁的135例病毒性肝炎患者。第一次在黄疸期,第二次在黄疸消失、主要的临床及实验室指标正常后检查。结果发现患者血清胡萝卜素含量明显增高,而维生素A含量却下降。在病程极期,血清胡萝卜素2.13±1.14毫克％;缓解期1.57±0.81毫克,健康人则为0.87±0.27毫克。(P<0.001)。血清维生素A含量在病程极期0.31±0.14毫克％;缓解期0.39±0.12毫克％,健康人0.51±0.10毫克％(P<0.001)。血清维生素A水平的降低,虽然部份可用胆汁分泌障碍、肠道吸收减弱来解释,但主要是肝脏损害影
The accumulation of vitamin A and the process of synthesizing vitamin A by carotene are all performed in the liver. Vitamin A enters various organs and tissues from the liver. Some data indicate that in the lack of vitamin A and lack of body immunity indicators - phagocytic index and complement titer - significantly reduced. Vitamin A can enhance the body's resistance to certain toxicants and toxins, and have anti-nutritional disorders. It activates insulin and promotes the storage of glycogen in the liver, and the tolerance of the liver to the causative agent depends on the degree of glycogen production in the liver. To investigate the dynamic changes of serum vitamin A and carotene levels in patients with viral hepatitis, the authors examined 135 patients with viral hepatitis aged 14-66 years. The first time in jaundice, the second disappeared in the jaundice, the main clinical and laboratory indicators of normal post-test. The results showed that patients with serum carotene increased significantly, while the vitamin A content decreased. The duration of the disease, serum carotenoids 2.13 ± 1.14 mg%; remission 1.57 ± 0.81 mg, healthy people were 0.87 ± 0.27 mg. (P <0.001). Serum vitamin A levels were 0.31 ± 0.14 mg% at the very onset of disease duration, 0.39 ± 0.12 mg% at remission, and 0.51 ± 0.10 mg% (P <0.001) of healthy individuals. Serum vitamin A levels decreased, although some may be bile secretion disorders, reduced intestinal absorption to explain, but mainly liver damage shadow