【摘 要】
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Objective This study was conducted to investigate whether the over-expression of anti-inflammation factor IL-10 can induce pulmonary fibrosis(PF) by activating JAK2/STAT3 pathway phosphorylation and t
【机 构】
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Key Laboratory of Tumour Molecular Biology in Binzhou Medical University, Department of Biochemistry
【出 处】
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中华医学会呼吸病学年会——2013第十四次全国呼吸病学学术会议
论文部分内容阅读
Objective This study was conducted to investigate whether the over-expression of anti-inflammation factor IL-10 can induce pulmonary fibrosis(PF) by activating JAK2/STAT3 pathway phosphorylation and thereby pro mote the expression of such down-stream target as the apoptotic protein Bax.IL-10 up-stream regulated micro RNA, miR-98 was also investigated.
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