ANG Ⅱ INDUCES CAPILLARY FORMATION FROM ENDOTHELIAL CELLS VIA THE AT1R-DEPENDENT INOSITOL REQUIRING E

来源 :第二十四届长城国际心脏病学会议、亚太心脏大会暨国际心血管病预防与康复会议(The 24th Great Wall Int | 被引量 : 0次 | 上传用户：NickFlanders

【摘要】

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【作者】

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Wang Xiang Yong-Ping Bai Dan Hong Hai-Chao Gao Ling-Fang Li Ling-Ping Zhu Quan Sun Guo-Gang Zhang

【机构】

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Department of Cardiovascular Medicine,Xiangya Hospital,Central South University,Changsha 410078,Chin

【出处】

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第二十四届长城国际心脏病学会议、亚太心脏大会暨国际心血管病预防与康复会议(The 24th Great Wall Int

【发表日期】

：

2013年2期

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Objectives Previous studies have demonstrated an important interaction between angiotension Ⅱ type 1 receptor (AT1R) and angiotension Ⅱ (Ang Ⅱ)-induced capillary formation from endothelial cells and vascular endothelial growth factor (VEGF).However, the underlying mechanism remains elusive.Recent studies revealed that the unfolded protein response regulates an angiogenic response by the kidney epithelium during ischaemic stress.

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ANG Ⅱ INDUCES CAPILLARY FORMATION FROM ENDOTHELIAL CELLS VIA THE AT1R-DEPENDENT INOSITOL REQUIRING E

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