危重新生儿肾上腺皮质功能的临床研究

来源 :中国妇幼保健 | 被引量 : 0次 | 上传用户:garry0809
下载到本地 , 更方便阅读
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
目的:探讨危重新生儿肾上腺皮质功能状态与病情严重程度的关系及促肾上腺皮质激素(ACTH)刺激试验对肾上腺皮质功能不全患儿预后的作用。方法:根据新生儿危重病例评分法(NCIS),将2011年10月~2012年11月石河子大学医学院第一附属医院收治的34周≤胎龄﹤42周、日龄<2天的60例新生儿分为非危重组、轻度危重组和重度危重组,采用化学发光免疫技术检测患儿皮质醇水平;根据皮质醇水平将危重患儿分为肾上腺皮质功能不全(AI)组和肾上腺皮质功能正常(AN)组;对危重患儿中合并AI者进行小剂量ACTH刺激试验,观察皮质醇水平及血压、血糖、血钾和血钠水平的变化。结果:非危重组、轻度危重组和重度危重组皮质醇水平分别为(12.82±3.96)μg/dL、(20.72±10.65)μg/dL和(21.67±8.58)μg/dL,轻度危重组和重度危重组皮质醇水平高于非危重组(P<0.05);轻度危重组与重度危重组皮质醇水平比较差异无统计学意义(P>0.05);轻度危重组和重度危重组AI的发生率分别为28.57%和47.37%,二者比较差异有统计学意义(P<0.05);危重患儿中,AI组的皮质醇水平、收缩压和血糖水平低于AN组(P<0.05),血钾和血钠水平比较差异无统计学意义(P>0.05);合并AI的患儿中,小剂量ACTH刺激试验后的皮质醇水平、收缩压和血糖水平较刺激试验前升高,血钾和血钠水平比较差异无统计学意义(P>0.05)。结论:危重新生儿肾上腺皮质功能状态与病情严重程度有关,皮质醇水平在轻度危重症时升高,当病情进一步加重时却未相应升高;危重新生儿合并AI时血压及血糖水平偏低;ACTH刺激能有效升高皮质醇、收缩压及血糖水平,具有一定的治疗作用。 Objective: To investigate the relationship between functional status of adrenal cortex and severity of critically ill newborn infants and the effect of adrenocorticotrophic hormone (ACTH) stimulation test on the prognosis of children with adrenal insufficiency. Methods: According to the Neonatal Critical Illness Scale (NCIS), 60 patients with gestational age <42 weeks and days <2 days 34 weeks after birth in the First Affiliated Hospital of Shihezi University from October 2011 to November 2012 were enrolled. Neonates were divided into non-critical group, mild-severe group and severe-severe group. The level of cortisol in children was detected by chemiluminescence immunoassay. According to the level of cortisol, the critically ill children were divided into adrenal insufficiency (AI) group and adrenal cortex (AN) group. In low-dose ACTH stimulation group, the patients with complicated AI in critically ill children were observed. The changes of cortisol and blood pressure, blood glucose, serum potassium and serum sodium were observed. Results: The levels of cortisol were (12.82 ± 3.96) μg / dL, (20.72 ± 10.65) μg / dL and (21.67 ± 8.58) μg / dL respectively in the non-critically ill group, mild- (P <0.05). There was no significant difference in the level of cortisol between mild and severe critically ill patients (P> 0.05). The levels of Cortisol in mild and severe critically ill patients were significantly higher than those in non-critically ill patients (P <0.05) (P <0.05). The incidence of cortisol, systolic blood pressure and blood glucose in AI group were significantly lower than those in AN group (P <0.05) ), Serum potassium and serum sodium levels were not significantly different (P> 0.05); cortisol levels, systolic blood pressure and blood glucose levels in low-dose ACTH stimulation test in children with AI were higher than those before stimulation test, Serum potassium and serum sodium levels were no significant difference (P> 0.05). Conclusions: The functional status of adrenocortical in critically ill neonates is related to the severity of the disease. Cortisol level is elevated in mild critically ill patients but not increased when the condition is further aggravated. Blood pressure and blood glucose levels are low in critically ill newborns with AI ; ACTH stimulation can effectively increase cortisol, systolic blood pressure and blood glucose levels, has a certain therapeutic effect.
其他文献
目的:新生儿难治性惊厥以治疗困难,预后欠佳成为临床诊治中的难点.有一部分难治性惊厥患儿病因不明,尝试对这类患儿进行基因检测,为临床上对该类疾病的病因诊断、评估预后提
会议
目的:将操作简便、结果易读、便于临床推广的振幅整合脑电图(aEEG)引入新生儿难治性惊厥患儿床旁脑功能的监测并评估其在难治性惊厥中的表现特点及应用价值.方法:对27例新生
会议
目的:对比研究极低出生体重儿与支气管肺发育不良患儿生后1年内下呼吸道感染临床特点.方法:回顾研究2010年1月~2012年1月于某医院新生儿科收治的确诊的VLBW患儿58例及BPD患儿9
会议
目的:探讨Crigler-Najjar综合征的临床特征及诊断.方法:分析1例经基因检测确诊为Crigler-Najjar综合征患儿的临床特征及其基因突变位点结果.结果:患儿于出生后5天发现黄疸,治
会议
本文介绍一例新生儿先天性肾发育不良,并检索文献进行分析,提出新生儿期发病的先天性肾发育不良预后不佳,所以如能在产前作出诊断减轻孕妇的心理负担,及时采取必要的措施是十分必
目的:探讨布地奈德(Budesonide)联合特布他林(Terbutaline)雾化吸入及前列腺素E1(Prostaglandin E1,PGE1)雾化吸入对高氧诱发新生鼠肺损伤的影响.方法:选用新生Wistar乳鼠120
会议
请下载后查看,本文暂不支持在线获取查看简介。 Please download to view, this article does not support online access to view profile.
随着科技的发达,人们的生活真是越来越舒适了。我们轻松地利用遥控器开启车库大门、电视机、录像机、DVD放映机,我们也能以无线电话、呼叫器、以PDA随时随地进行通讯联络。我
目的:为进一步分析小于胎龄儿天然免疫细胞分泌抗病毒因子能力低下的原因,分析了单核的数量以及肿瘤坏死因子表达,以及出生体重对单核抗病毒能力的影响.方法:收集来自四川大
会议