论文部分内容阅读
在孕12周内母体发生急性风疹常致胎儿感染,从治疗性流产胎儿组织中,病毒的分离率达90%,足月分娩的婴儿中81%有血清学感染的证据。在此期受感染的婴儿约85%有严重损害,胎儿感染是慢性的,病毒在咽部存在至出生后数周在敏感的接触者中可引起风疹,病毒在胎盘存留的时间尚不清楚。在12~16孕周母体患风疹中,曾发现胎儿感染率为54%,其中35%遭受某种程度的损害。妊娠后期母体患风疹的胎儿感染率至孕23~26周下降至25%左右,而在孕末的2个月又有升高。在孕16周后的胎儿感染
In the first 12 weeks of gestation, there is a common fetal infection of the mother with an acute rubella, and 90% of the virus is isolated from fetal abortion, and 81% of full-term infants have serologic evidence of infection. Approximately 85% of infected infants during this period are severely damaged. Fetal infections are chronic. Viruses can cause rubella in susceptible contacts during the throat phase up to several weeks after birth. The timing of the virus’s persistence in the placenta remains unclear. In 12 to 16 gestational weeks mother with rubella, it was found that the rate of fetal infection was 54%, of which 35% suffered some degree of damage. In the second trimester of pregnancy, the fetus infected with rubella has been reduced to about 25% of pregnant women by 23 to 26 weeks of gestation but rises again in the second trimester of pregnancy. Fetal infection after 16 weeks pregnant