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本实验在迷走神经切断和未切断的麻醉兔上观察比较了单独电刺激下丘脑室旁核(PVH)、下丘脑背内侧核(DMH)及合并刺激PVH与DMH所诱发的缺血性心电变化(ST偏移)、定性心律失常(室性早搏)及升压反应。结果提示:DMH是诱发缺血性心电变化的高反应区,PVH则是诱发室性心律失常、血压升高的高反应区,PVH与DMH合并刺激能使两者反应叠加。迷走神经完整组与迷走神经切断组的结果比较提示,迷走神经的存在使刺激DMH及PVH+DMH诱发的缺血性ST偏移减轻,表明迷走神经对心肌缺血有保护作用,但对诱发室性早搏及升压反应的影响不明显。
In this study, vagotomy and vaginal anesthesia in rabbits were observed and compared the electrical stimulation of the hypothalamic paraventricular nucleus (PVH), hypothalamic dorsal medial nucleus (DMH) and the combined stimulation of PVH and DMH induced ischemic changes (ST offset), arrhythmia (premature ventricular contractions) and ascending response. The results suggest that DMH is a high reaction area for inducing ischemic electrocardiographic changes and PVH is a high reaction area for inducing ventricular arrhythmia and blood pressure. Combined stimulation of PVH and DMH can make the two reactions superpose. Vagus nerve intact group and vagotomy group compared the results suggest that the presence of the vagus nerve to stimulate the DMH and PVH DMH-induced ischemic ST offset reduction, indicating that the vagus nerve has a protective effect on myocardial ischemia, but the induction of ventricular premature beats and l The impact of pressure reaction is not obvious.