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实验选用１５只新西兰兔，分为３组，每组５只，分别为颈椎病模型组，对照组及和颈椎病模型加人工麝香饲养组。在造模后８月时处死动物。颈椎病模型加人工麝香饲养组动物在处死前喂以人工麝香２周。取动物颈椎间盘组织，用生化方法测定其中组织胺、５－羟色胺、ＰＧＥ２及６－酮－ＰＧＦ１ａ水平，结果表明：（１）颈椎病模型动物颈椎间盘中四种炎症介质水平明显高于其正常对照组动物的水平。（２）人工麝香对退变颈椎间盘中的四种炎症介质水平具有明显降低作用。
In the experiment, 15 New Zealand rabbits were selected and divided into 3 groups with 5 rats in each group, which were respectively the cervical spondylosis model group, the control group, and the cervical spondylosis model plus the artificial musk-feeding group. Animals were sacrificed 8 months after modeling. Models of cervical spondylosis plus artificial muskets were fed with artificial musk for 2 weeks prior to sacrifice. Animals were harvested from cervical intervertebral discs and their histamine, 5-hydroxytryptamine, PGE2, and 6-keto-PGF1a levels were measured by biochemical methods. The results showed that: (1) The levels of four inflammatory mediators in cervical discs of cervical spondylosis model animals were significantly higher than normal levels. The level of animals in the control group. (2) Artificial musk significantly reduced the levels of four inflammatory mediators in degenerated cervical discs.