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用外源水杨酸（ｓａｌｉｃｙｌｉｃ ａｃｉｄ，ＳＡ）处理烟草（ Ｎｉｃｏｔｉａｎａｔａｂａｃｕｍ Ｌ．） 培养细胞，证实ＳＡ能诱导脂质过氧化，抑制过氧化氢酶活性并诱导ＰＲ＿１ 基因的表达。对苯二酚和Ｈ２Ｏ２ 也能不同程度地诱导烟草培养细胞的脂质过氧化和ＰＲ＿１ 基因的表达，但不能抑制过氧化氢酶活性。ｍＲＮＡ 和蛋白合成抑制剂（ 鹅膏覃碱和环己亚胺） 不能诱导烟草培养细胞的脂质过氧化和ＰＲ＿１ 基因的表达，但与ＳＡ协同作用能更有效地诱导烟草培养细胞的脂质过氧化，但强烈地抑制ＳＡ诱导的ＰＲ＿１ 基因的表达。因而推测，ＳＡ或其他诱导物可能通过和过氧化氢酶或植物体内其他大分子相互作用产生活性代谢物或自由基，诱导脂质过氧化，进而激活抗性基因的表达。实验结果表明，过氧化脂质分子在ＳＡ信号传导途径中扮演重要角色
Cultured cells of tobacco (Nicotiana tabacum L.) treated with salicylic acid (SA) demonstrated that SA can induce lipid peroxidation, inhibit catalase activity and induce PR 1 gene expression. Hydroquinone and H2O2 also induced lipid peroxidation and PR1 gene expression in tobacco cultured cells to varying degrees, but not catalase activity. Inhibitors of mRNA and protein synthesis (aqin and cycloheximide) failed to induce lipid peroxidation and PR1 gene expression in tobacco cultured cells, but synergistically with SA could more effectively induce lipid peroxidation in cultured tobacco cells Oxidation, but strongly inhibits the SA-induced expression of PR 1 gene. Thus, it is speculated that SA or other inducers may induce lipid peroxidation, which in turn may activate the expression of resistant genes by interacting with catalase or other macromolecules in the plant to produce active metabolites or free radicals. The experimental results show that lipid peroxidation molecules play an important role in the SA signal transduction pathway