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患儿,男,5岁.其父自1995年5月起在某厂从事黄丹粉碎、包装至今,下班后穿工作服回家.患儿常穿着他父亲的工作服和鞋玩耍,玩后常不洗手便进食.1996年3月,患儿出现头痛、乏力、食欲不振、腹痛、稀便,就诊于某医院,给予对症治疗无明显效果.5月12日,患儿腹痛加剧,其父猛然想起防疫站工作人员所讲的铅中毒知识,便带患儿来我站就诊.查体:T 36.8℃、P 96次/分、R23次/分、BP 9/6kPa,腹部平软,肝脾未触及,牙龈“铅线”明显,呈贫血貌.询问得知有铅接触史.化验尿铅0.083mg/L,诊为铅中毒.开始驱铅,给予CaNa_2EDTA0.25g加入10%葡萄糖300mg/l静滴.共驱铅治疗 3个疗程,症状明显改善,验尿铅 0.02mg/L.半年后来我站复查,以上症状消失,
Children, male, aged 5. His father since May 1995 Huangdan in a mill crush, packaging so far, after get off work to wear overalls home children often wear his father’s work clothes and shoes to play, often after playing In March 1996, children with headache, fatigue, loss of appetite, abdominal pain, loose stools, visited a hospital, giving symptomatic treatment no significant effect on May 12, children with abdominal pain intensified, and his father suddenly remembered Epidemic prevention station staff talking about lead poisoning knowledge, they bring children to my station .Check the body: T 36.8 ℃, P 96 beats / min, R23 beats / min, BP 9 / 6kPa, abdominal soft, liver and spleen not Touched, gingival “lead line” was significantly anemic appearance. Asked to know the history of lead exposure. Urinary lead test 0.083mg / L, diagnosed as lead poisoning began to drive lead, given CaNa_2EDTA0.25g added 10% glucose 300mg / l static Drip. A total of three courses of lead treatment, the symptoms were significantly improved, urine test lead 0.02mg / L. Six months later I check the station, the above symptoms disappear,