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本文以34只家兔对失血性休克2小时心肌进行了组织学及组织化学观察。实验分三组:正常组、实验对照组和休克组。结果表明:失血性休克时,心肌细胞形态和线粒体定位发生很大变化,细胞肿胀,线粒体移位并且积聚脱失相混杂。经显微分光光度计定量分析表明,乳酸脱氢酶(LDH)活性有所提高,但与对照组比无显著差异,而琥珀酸脱氢酶(SDH)活性显著下降(P<0.05)。因此本文认为,能量产生减少,能源危机可能是失血性休克死亡的主要因素之一。
In this paper, 34 rabbits hemorrhagic shock 2 hours of myocardial tissue histological and histological observations. The experiment was divided into three groups: normal group, experimental control group and shock group. The results showed that during the process of hemorrhagic shock, the morphology and mitochondrial localization of cardiomyocytes changed greatly. The cells swollen and mitochondria translocated, and the accumulation and loss were mixed. Quantitative analysis by micro-spectrophotometer showed that lactate dehydrogenase (LDH) activity increased, but no significant difference compared with the control group, while succinate dehydrogenase (SDH) activity decreased significantly (P <0.05). Therefore, this paper believes that the reduction of energy production, energy crisis may be one of the major factors in the death of hemorrhagic shock.