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日本研究人员发现一种蛋白质分子,可使具有抗病毒作用的Ⅰ型干扰素分泌量增加。这一成果将有望使人为增强肌体的抗病毒反应成为可能。日本理化研究所日前发布新闻公报说,当病毒侵入人体时,免疫系统中的Toll样受体会识别出病毒,并向浆细胞样树突状细胞发送信号,接收到信号的浆细胞样树突状细胞会产生Ⅰ型干扰素抗病毒。公报说,浆细胞样树突状细胞产生Ⅰ型干扰素的具体机制目前尚不清楚,但一种名为PDC-TREM的蛋白质分子,只在浆细胞样树突状细胞接收到Toll样受体发出的信号后才会表达。
Japanese researchers found that a protein molecule, can make antiviral effect of type I interferon secretion increased. This result is expected to make artificially enhance the body's antiviral response possible. Japan's National Institute of Science and Technology recently released a press release saying that when a virus invades the body, Toll-like receptors in the immune system recognize the virus and send signals to plasmacytoid dendritic cells that receive the plasmacytoid dendrites Cells produce type 1 interferon antivirals. The communiqué said that the specific mechanism by which plasmacytoid dendritic cells produce type I interferons is not yet clear, but a protein molecule called PDC-TREM, which receives Toll-like receptors only from plasmacytoid dendritic cells After the signal sent out will be expressed.