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1.通过对440名潜水员的调查,发现饮酒的潜水员急性减压病的发病率明显低于不饮酒者。 2.通过对127名潜水员的调查发现,饮酒的潜水员减压性骨坏死的发病率明显低于不饮酒者。 3.在49名减压性骨坏死患者中,不饮酒者的发病率是饮酒者的3倍。 4.18名潜水员在9～29年来饮酒与非饮酒期间减压病的发病情况作自身对照,他们共患急性减压病31次,减压性骨坏死5人。均发生于非饮酒期。 5.乙醇治疗家兔急性减压病的实验指出,乙醇对家兔急性减压病的治疗是有效的。治疗组与对照组家兔死亡差别极显著。 6.乙醇治疗减压病的机理是降低气泡的表面张力;影响血小板的分泌、粘附、聚集和促凝功能;促进体内氮气和二氧化碳气体的排出,增加血输出量,改善微循环等。
1. According to a survey of 440 divers, the incidence of acute decompression sickness among drinking divers was significantly lower than that of non-drinkers. 2. Through the survey of 127 divers, the incidence of decompressive osteonecrosis of drinkers was significantly lower than that of non-drinkers. 3. Among the 49 patients with decompressive osteonecrosis, the incidence of non-drinkers was three times higher than that of drinkers. 4.18 divers in the 9 to 29 years of drinking and non-drinking during the onset of decompression sickness as a self-control, they co-suffering from acute decompression sickness 31 times, 5 patients with decompressive osteonecrosis. Occurred in non-drinking period. Ethanol treatment of acute decompression sickness in rabbits pointed out that ethanol treatment of acute decompression sickness in rabbits is effective. The difference between the treatment group and the control group was extremely significant. 6. Ethanol treatment of decompression sickness mechanism is to reduce the surface tension of air bubbles; affect the platelet secretion, adhesion, aggregation and coagulation; promote the body’s nitrogen and carbon dioxide gas emissions, increase blood output, improve microcirculation.