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本文以35只家兔分三组对失血性休克2小时动物心肌进行了三种方法的组织化学观察。结果看到:失血性休克发生后,心肌儿茶酚胺(CA)含量明显减弱以至耗竭(乙醛酸诱发CA荧光方法)、心肌糖原(PAS反应)和磷酸化酶(磷酸化酶改良法)大量脱失,部分心肌细胞明显肿胀。而假手术对上述指标影响很轻。结果说明,失血性休克对心肌组织化学影响很大,导致心脏交感神经功能低下,心肌代谢紊乱,可能是造成本文绝大部分(13/14)休克动物死亡的主要因素。
In this paper, 35 rabbits were divided into three groups on hemorrhagic shock 2 hours animal myocardium were three methods of histochemical observation. The results showed that after the onset of hemorrhagic shock, the content of myocardial catecholamine (CA) was significantly weakened and even depleted (glyoxylate induced CA fluorescence method), myocardial glycogen (PAS reaction) and phosphorylase (phosphorylase improved method) Loss, some myocardial cells significantly swollen. Sham surgery on the above indicators have a mild effect. The results show that hemorrhagic shock has a great influence on myocardial histochemistry, leading to cardiac sympathetic dysfunction and myocardial metabolic disorders, which may be the main factors contributing to the death of most (13/14) shock animals in this study.