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病毒感染能活化凝血系统和内皮上的单核细胞受体，从而增强单核细胞的粘附；还可能改变细胞脂质代谢，促使血液凝固和细胞转化。与动脉粥样硬化（ＡＳ）发生发展有密切关系。
Viral infection activates the monocyte receptors on the coagulation system and the endothelium, thereby enhancing monocyte adhesion; it may also alter cellular lipid metabolism, promote blood coagulation and cellular transformation. And atherosclerosis (AS) is closely related to the occurrence and development.