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一、引言慢性苯中毒的血液毒性已知道近一个世纪。苯的毒性效应包括白细胞减少、血小板减少、贫血、暂时性白细胞增多、淋巴细胞减少,偶见淋巴细胞增多,罕见假性Pelger—Hu et氏白细胞抗渗透性异常,粒细胞吞噬功能减低,嗜中性细胞糖元含量减少和过氧化物酶活性抑制,酸性磷酸酶和β—葡萄糖苷酸酶活性增高,碱性磷酸酶、髓过氧化物酶和脂质含量增高。还见Ea和E_(18)玫瑰花
I. Introduction Hematotoxicity of chronic benzene poisoning has been known for nearly a century. The toxic effects of benzene include leucopenia, thrombocytopenia, anemia, transient leukocytosis, lymphopenia, rare lymphocytosis, rare pseudo-Pelger-Hu et al leukocyte anti-permeability abnormalities, decreased granulocyte phagocytosis, Decreased glycogen content and peroxidase activity, increased the activity of acid phosphatase and β-glucuronidase, increased the content of alkaline phosphatase, myeloperoxidase and lipid. See also Ea and E_ (18) roses