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早在70年代国外学者提出血管内皮损伤是动脉粥样硬化(AS)形成的重要始动环节,随后研究发现内皮细胞合成、分泌的一氧化氮(NO)不仅能调节血管平滑肌张力,还具有抑制血小板、中性粒细胞在血管内膜的粘附、聚集和激活,抑制血管平滑肌细胞增殖。机体存在一种调节NO合成的内
As early as the 1970s, foreign scholars proposed that vascular endothelial injury is an important starting point in the formation of atherosclerosis (AS). Subsequently, it was found that the synthesis and secretion of nitric oxide (NO) by endothelial cells not only regulated the vascular smooth muscle tone but also inhibited Platelets, neutrophils in the intima of the adhesion, aggregation and activation, inhibition of vascular smooth muscle cell proliferation. There is a body that regulates NO synthesis inside