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以往的工作表明大肠杆菌内毒素休克动物肺循环阻力升高,山茛菪碱(654-2)治疗可使其缓解。为进一步探讨内毒素致肺血管收缩作用的部位并与一些血管活性物质的作用进行
Previous work showed that E. coli endotoxin shock animals with increased pulmonary vascular resistance, anisodamine (654-2) treatment can make it alleviate. To further explore the role of endotoxin induced pulmonary vasoconstriction and the role of some vasoactive substances