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急性心肌梗死(AMI)后的无复流、心律失常、心室重构是AMI患者愈后的重要指标。而神经内分泌的激活、免疫活动的异常等多种机制参与了AMI时的病理生理过程~([1,2]),国内外学者相继发现心脏肾上腺素受体的自身抗体可引发心脏结构和功能的改变,可能参与AMI后的无复流、心律失常、心室重构过程。通过研究急性ST段抬高心肌梗死(STEMI)直接冠状动脉介入治疗(PCI)出现无复流的患者,其PCI前后β1、
Acute myocardial infarction (AMI) after no-reflow, arrhythmia, ventricular remodeling AMI patients is an important indicator of prognosis. Neuroendocrine activation, abnormal immune activity and other mechanisms involved in the pathophysiology of AMI ~ ([1,2]), domestic and foreign scholars have found that cardiac adrenergic receptor autoantibodies can lead to cardiac structure and function Changes may be involved in AMI after no reflow, arrhythmia, ventricular remodeling process. By studying the patients with no-reflow after direct coronary intervention (PCI) in acute ST-segment elevation myocardial infarction (STEMI), the levels of β1,