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晚期血吸虫病由于肝脏逐渐地形成了硬化,演变至肝机能严重减损,而丧失了代偿作用;因之,血吸虫病本身的重要性乃由肝硬化所取代,并逐步走向衰竭.肝脏衰竭的临床表现为腹水形成、黄疸出现和无其他原因的发热.后二者在晚期血吸虫病肝硬化患者中虽非常见,但亦非罕见.患者腹腔内腹水蓄积与门静脉高压和血浆胶性渗透压减低这两种因素的联合作用有重要关系.在腹水开始蓄积时,其他促成钠与水潴留的机制亦参与了作用,其中以肾上腺皮质的储钠激素醛固酮的增加具有密
Advanced schistosomiasis gradually hardens due to the liver and evolves into a severe impairment of liver function, losing its compensatory effect; therefore, the importance of schistosomiasis itself is replaced by cirrhosis and progressively depleted. Clinical failure of liver failure Manifested as the formation of ascites, jaundice and no other causes of fever.The latter two are uncommon in patients with advanced schistosomiasis cirrhosis, but not uncommon.Patients with intraperitoneal ascites accumulation and portal hypertension and plasma osmolarity decreased There is an important relationship between the combined effects of the two factors.Other mechanisms contributing to sodium and water retention also play a role in the initiation of ascites accumulation, with an increase in the adrenocortical sodium aldosterone aldosterone