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临床研究证实，血管紧张素转换酶抑制剂能治疗和预防心衰，已知由特发性扩张性心肌病导致的晚期心衰患者，其心肌a1受体密度下降，而G；。蛋白（抑制性GTP结合蛋白的a亚基）含量增加。为探讨卡托普利防治心衰作用机理，本文研究了卡托普利对心衰患者心肌a受体和G；。蛋白含量的
Clinical studies have confirmed that angiotensin-converting enzyme inhibitors can treat and prevent heart failure, and are known to have decreased myocardial A1 receptor density in patients with advanced heart failure due to idiopathic dilated cardiomyopathy. The protein (a subunit of inhibitory GTP-binding protein) is increased in content. In order to explore the mechanism of action of captopril in preventing and treating heart failure, this paper studied the effects of captopril on cardiac a receptor and G in patients with heart failure. Protein content