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急性心肌梗死(AMI)发生后,患者的病情将逐渐向慢性充血性心力衰竭(CHF)发展,此时体内的肾素-血管紧张素-醛固酮系统(RAAS)被激活,增加了水钠潴留和周围血管阻力,导致心室进行性扩张,最终加重心力衰竭,因此,阻断RAAS、减少血管紧张素Ⅱ(AngⅡ)的产生,对治疗CHF极为重要。本文比
After an acute myocardial infarction (AMI), the patient’s condition progresses to chronic congestive heart failure (CHF), when the body’s renin-angiotensin-aldosterone system (RAAS) is activated, increasing sodium and water retention Peripheral vascular resistance, leading to progressive ventricular dilatation, and ultimately aggravate heart failure, therefore, block RAAS, reduce the production of angiotensin (Ang Ⅱ), for the treatment of CHF is extremely important. This article than