【摘 要】
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支气管哮喘(asthma,简称哮喘)是一种以嗜酸性粒细胞、嗜中性粒细胞和淋巴细胞浸润等多炎症细胞、炎症介质和细胞因子共同参与的气道慢性非特异性炎症性疾病,特征为黏液高分泌
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支气管哮喘(asthma,简称哮喘)是一种以嗜酸性粒细胞、嗜中性粒细胞和淋巴细胞浸润等多炎症细胞、炎症介质和细胞因子共同参与的气道慢性非特异性炎症性疾病,特征为黏液高分泌和气道高反应性。尽管在哮喘发病机制中有各种假说,如变应原假说、饮食假说及卫生假说[1],都是设想哮喘在环境中的发病机理,但是仍以气道炎症学说得到医学界广泛关注与证实,研究表明:炎症细胞的释放、趋化是气道炎症形成的基础,
Bronchial asthma (asthma) is a chronic, nonspecific airway inflammatory disease involving multiple inflammatory cells such as eosinophils, neutrophils and lymphocyte infiltration, inflammatory mediators and cytokines, and is characterized by Mucus hypersecretion and airway hyperresponsiveness. Although various hypotheses such as allergen hypothesis, dietary hypothesis and hygienic hypothesis in the pathogenesis of asthma [1] have assumed the pathogenesis of asthma in the environment, they are still widely concerned by the medical theory of airway inflammation Confirmed that studies show that: the release of inflammatory cells, chemotaxis is the basis for the formation of airway inflammation,
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