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血小板在脑梗死发病前、发病过程及病后都发生变化。血小板计数和体积改变可能是脑梗死形成的一个原因,而不是梗死后继发改变。在脑梗死患者急性期检测血小板参数可作为脑梗死加重的一个危险因素进行观察,同时可作为抗血小板治疗效果的观察指标。血小板的异常变化,与梗死范围的扩展和病情的预后有关,病情越重,梗死面积越大,血小板参数改变越明显。血小板计数减少、平均血小板体积及血小板分布宽度增加是与脑梗死病情相关的病理生理学基础。抗血小板药物能够抗血小板黏附性和聚集性,防止血栓形成,有助于防止动脉粥样硬化和心肌梗死。
Platelet before the onset of cerebral infarction, the incidence of disease and disease have changed. Platelet counts and volume changes may be a cause of cerebral infarction, rather than secondary changes after infarction. The detection of platelet parameters in the acute phase of cerebral infarction can be used as a risk factor for aggravating cerebral infarction, and can be used as an indicator of antiplatelet therapy. Abnormal platelet changes, and the expansion of the infarct range and the prognosis of the disease, the more serious the disease, the greater the infarct size, the more obvious changes in platelet parameters. Reduced platelet counts, mean platelet volume, and increased platelet distribution are the pathophysiological bases associated with the condition of the cerebral infarction. Anti-platelet drugs can prevent platelet adhesion and aggregation, prevent thrombosis and help prevent atherosclerosis and myocardial infarction.