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近年来,许多作者进行了大量实验研究,证明了某些遗传性溶血性贫血,特别是遗传性球形红细胞增多症的发病原因,是由于红细胞膜旦白异常。这些实验,一部份通过对可溶性膜旦白质的研究显示膜旦白质结构异常;一部份是研究其功能紊乱,并着重研究膜的转化及膜旦白磷酸化作用。
In recent years, many authors conducted a large number of experimental studies to prove that some hereditary hemolytic anemia, in particular, is the cause of genetic hereditary polycythemia is due to abnormal erythrocyte membrane protein. Some of these experiments, through studies on soluble membrane proteins, revealed an abnormal structure of membrane proteins, partly as a result of studies on their dysfunctions and their emphasis on membrane transformation and membrane phosphorylation.