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目的 观察血糖增高对急性脑梗死灶容积、临床神经功能缺损程度及死亡率的影响。方法 经明确诊断的急性脑梗死病人149例,分为高血糖观察组52例,而血糖正常的97例为对照组,对脑梗死灶容积大小,临床神经功能缺损程度(入院时及治疗1个月后)的变化和治疗一个月时的死亡率进行观察对比。结果 观察组患者脑梗死灶容积平均为(2 93±0 52)cm3,而对照组脑梗死灶容积平均为(2 11±0 44)cm3(P<0 01);观察组早期临床神经功能缺损程度平均为(28 14±9 13)分;对照组平均为(19 76±9 16)分(P<0 01);观察组治疗 1 个月存活者临床神经功能缺损程度平均为(19 11±9 11)分;对照组平均为(11 93±8 97)分(P<0 01);观察组治疗1个月后死亡率为36 5%,对照组为17 5%(P<0 05)。结论 高血糖可造成脑梗死容积增大,加重临床神经功能缺损程度并影响病情恢复,加重后遗症、增加死亡率。故应尽早判断,积极治疗。
Objective To observe the effect of hyperglycemia on the volume of acute cerebral infarction, the degree of clinical neurological deficit and mortality. Methods A total of 149 patients with acute cerebral infarction diagnosed were divided into hyperglycemia observation group (n = 52) and hyperglycemia observation group (n = 97) as control group. The volume of cerebral infarction volume, the degree of clinical neurological deficit Month) changes and treatment of one month after the mortality observed and compared. Results The volume of cerebral infarction in observation group was (2 93 ± 0 52) cm 3, while that in control group was (2 11 ± 0 44) cm 3 (P 0 01). The early clinical neurological deficit (28 14 ± 9 13) points in the control group and (19 76 ± 9 16) points in the control group (P0 01). The average degree of clinical neurological deficits in the observation group at 1 month after treatment was (19 11 ± (11 93 ± 8 97) points in the control group (P <0.01). The mortality rate in the observation group after one month treatment was 36.5% and in the control group was 17 5% (P0.05) . Conclusions Hyperglycemia can increase the volume of cerebral infarction, aggravate the degree of clinical neurological deficit and affect the condition recovery, aggravate sequelae and increase the mortality rate. It should be judged as soon as possible, active treatment.