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幽门螺杆菌 ( Helicobacter Pylori,HP)感染作为慢性胃炎和消化性溃疡的重要致病因素已得到大量研究证实。HP通过细胞毒素、局部细胞免疫、激活氧自由基等多种渠道损伤胃粘膜[1] 。作者研究观察了 HP感染时胃窦粘膜及空腹血中胃泌素、生长抑素含量及 HP清除前后的变化 ,进一步
Infection of Helicobacter Pylori (HP) has been confirmed by a large number of studies as an important causative agent of chronic gastritis and peptic ulcer. HP through the cytotoxin, local cellular immunity, activation of oxygen free radicals and other channels damage gastric mucosa [1]. The authors studied the change of gastrin, somatostatin content in gastric mucosa and fasting blood before and after HP infection, and the changes before and after HP clearance