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传统学说肝硬化腹水的发生机制有:肝硬化引起门静脉血液回流障碍,使门静脉压力逐渐升高,静水压上升,肝功能损害引起血浆自蛋白减少,导致血浆胶体渗透压下降。肝硬化引起窦后梗阻,形成大盆肝淋巴液,超过胸导管的输送能力,自肝表面溢入腹腔。正常每日形成的肝淋巴液金为1。。。~15ooml,肝硬化者可增加到每日吕~IOL,如出现腹水可高达201才。由于体滚平衡失调,机体有效血容t减少,肾脏血液灌注量下降,
The traditional theory of cirrhosis of the ascites pathogenesis are: liver cirrhosis caused by portal vein reflux disorders, portal pressure gradually increased hydrostatic pressure, liver damage caused by plasma protein decreased, leading to decreased plasma colloid osmotic pressure. Cirrhosis caused sinus obstruction, the formation of large basin of liver lymphatic fluid over thoracic duct transport capacity, overflow from the liver surface into the abdominal cavity. The normal liver lymphatic fluid formed daily is 1. . . ~ 15ooml, cirrhosis can increase to daily Lu ~ IOL, ascites up to 201 before. As body roll balance disorders, the body reduces effective t blood flow, decreased renal blood perfusion,