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由于促使肝硬化从代偿期向失代偿期发展的因素尚不十分清楚，给肝硬化治疗带来了一定的困难。肝硬化失代偿期的主要病理机制是肝细胞功能障碍、广泛的侧枝循环形成以及肝纤维化。研究表明，一氧化氮（ＮＯ）对肝细胞有损害作用，可加重门脉血流动力学紊乱［１］，同时发现某些
The factors that promote the development of cirrhosis from decompensation to decompensation are not yet clear, which brings certain difficulties to the treatment of cirrhosis. The main pathologic mechanisms of decompensation of cirrhosis are hepatocellular dysfunction, extensive collateral circulation formation, and hepatic fibrosis. Studies have shown that nitric oxide (NO) has a detrimental effect on liver cells, can aggravate portal hemodynamic disorders [1], and found that some