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50年代在日本爆发了震惊世界的“水俣病”,经证明为甲基汞污染所致。环境中低剂量汞长期作用人体产生的遗传损伤问题。各国学者从不同角度,使用不同方法进行了一系列研究,但甲基汞对小鼠骨髓细胞SCE作用的研究尚未见报导,本文将这一实验研究报道如下:1 材料和方法 1.1 动物和剂量分组 采用本校实验动物部提供的小鼠,体重为23～26克,实验前观察3天,均健康无异常,随机分为实验组和对照组,每组10只,
Minamata disease that shocked the world broke out in Japan in the 1950s and proved to be caused by methylmercury pollution. Long-term effects of low doses of mercury in the environment on the genetic damage caused by the human body. Scholars from various countries from different angles, using a variety of methods conducted a series of studies, but methylmercury on the role of SCE mouse bone marrow cells have not been reported, this article will report the experimental study is as follows: 1 Materials and methods 1.1 animal and dose groups The mice provided by the experimental animal department of our school, weighing 23 ~ 26 grams, observed for 3 days before the experiment, were healthy and no abnormalities were randomly divided into experimental group and control group, 10 in each group,