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以Ｎ－甲基－Ｎ’－硝基－Ｎ－亚硝基胍（ＭＮＮＧ）诱发大鼠胃癌的方法观察胃炎消防治胃癌的作用。结果表明，胃炎消组肿瘤发生率（７／２０，３５％）明显低于空白对照组（１３／２２，５９．１％），且肿瘤只限于胃腺癌的早期癌，而空白组除出现胃腺癌的早期癌外，还出现了胃腺癌的晚期癌，并兼有十二指肠癌、胃平滑肌肉瘤和腹腔肿瘤。对两组胃和十二指肠癌变过程组织病理学进行计量分析，空白组平均分值４．１，胃炎消组平均分值２．７（Ｐ＜０．０５），说明胃炎消阻碍了ＭＮＮＧ引起组织癌变的病理进程
The role of gastritis in the treatment of gastric cancer was investigated with N-methyl-N’-nitro-N-nitrosoguanidine (MNNG)-induced gastric cancer in rats. The results showed that the incidence of tumor in the gastritis elimination group (7/20, 35%) was significantly lower than that in the blank control group (13/22, 59.1%), and the tumor was limited to the early cancer of gastric adenocarcinoma, while the blank group had no gastric gland In addition to early cancers, advanced cancers of gastric adenocarcinoma have also emerged, as well as duodenal cancer, gastric leiomyosarcoma and abdominal cavity tumors. The histopathology of the gastric and duodenal carcinogenesis of the two groups was analyzed quantitatively. The mean score of the blank group was 4.1, and the mean score of the gastritis elimination group was 2.7 (P<0.05), indicating that the gastritis elimination hindered the MNNG. The pathological process that causes cancerous tissue