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在中枢神经系统及胃肠道 ,一氧化氮合酶 (ＮＯＳ)分布广泛 ,它催化Ｌ -精氨酸形成具有高度生物活性的一氧化氮(ＮＯ) [1 ] 。研究表明 ,ＮＯ作为血管内皮衍生性舒张因子及特殊的神经递质 ,与胃粘膜保护及胃溃疡修复之间存在着密切联系。因此本实验通过束缚 -冷冻造成小鼠应激性
In the central nervous system and gastrointestinal tract, nitric oxide synthase (NOS) is widely distributed, which catalyzes the formation of L-arginine with high biological activity of nitric oxide (NO) [1]. Studies have shown that NO as a vascular endothelial-derived relaxing factor and special neurotransmitters are closely related to the protection of gastric mucosa and the repair of gastric ulcers. Therefore, in this experiment, mouse stress was caused by binding-freezing.