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肾综合征出血热（hemorrhaglcfeverwithrenalsyn－drome，HFRS）的病原虽已明确，但其发病机理还不十分清楚，许多研究发现HFRS患者血清总lgM，IgG等免疫球蛋白增高，尤以lgM增高最显著，并出现特异性的HFRS病毒抗体．补体传统激活途径增强而致血管CI，C3下降，C3裂解产物C3C增
Hemorrhagic fever with renal syndrome (HFRS) pathogen has been clear, but its pathogenesis is not yet clear, many studies found that HFRS patients serum lgM, IgG and other immunoglobulins increased, especially lgM increased most significantly, And the emergence of specific antibodies to HFRS virus. Enrichment of traditional ways of complement activation caused by blood vessels CI, C3 decreased C3 C C3 lysate increased