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动物实验研究表明，维生素Ｅ具有抗肝纤维化作用［１］，但具体机制不明。目前研究认为，贮脂细胞在肝纤维化形成中起关键作用。为探讨维生素Ｅ抗肝纤维化的细胞学机制，我们以白介素２作为刺激因素研究了维生素Ｅ对大鼠贮脂细胞增殖和胶原合成的作用。材料与方法一、材料（
Animal experiments show that vitamin E has anti-liver fibrosis [1], but the specific mechanism is unknown. The current study suggests that the storage of fat cells play a key role in the formation of liver fibrosis. To investigate the cytological mechanism of vitamin E against hepatic fibrosis, we investigated the effect of vitamin E on the proliferation and collagen synthesis of the lipid-storing cells in rats by using interleukin-2 as stimulating factor. Materials and methods First, the material (