论文部分内容阅读
职业倦怠作为当前社会从业人员普遍存在的心理现象,其生理机制主要与长期过度应激引起神经系统的紊乱相关,焦虑与职业倦怠关系密切,都受到神经系统和内分泌系统共同调节。神经系统中神经递质及其受体:如5-羟色胺,去甲肾上腺素和多巴胺的异常,导致焦虑的发生,可能诱发了职业倦怠。本文从激素水平、分子水平、基因水平三个方面分别阐述了职业倦怠发生和焦虑发生的机制以及两者之间相互作用的生物原理,为今后职业倦怠的心理预防以及基因治疗提供了生物医学研究相关的基础理论,也为职业倦怠易感人群,尤其是女性易感人群的焦虑情绪产生的机制研究奠定了详尽的生物医学方面的基础。
Occupational burnout, as a psychological phenomenon common to current social workers, is mainly related to nervous system disorders caused by long-term excessive stress. Anxiety and job burnout are closely related and are regulated by both the nervous system and the endocrine system. Neurotransmitters and their receptors in the nervous system: Abnormalities in serotonin, norepinephrine, and dopamine cause anxiety and may induce job burnout. In this paper, the mechanisms of occupational burnout and anxiety are described respectively from three aspects of hormone level, molecular level and gene level, and the biological principles of the interaction between the two are provided, which provide a biomedical research for the future psychological burnout prevention and gene therapy The related basic theory also lays a detailed biomedical basis for the study of the mechanism of anxiety induced by the occupational burnout susceptible population, especially the female susceptible population.