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患者男,59岁。1991年7月1日因发作性心前区痛,伴胸闷3天,活动后加重就诊。曾有冠心病史。听诊:心音低,无杂音,律整,血压14/kPa,即做心电图:T V_1浅倒,TV_(2-4)正负双向(正向<负向),S-T均呈上斜形,略上抬,V_(2-4)U波深倒,达0.2～0.4mv,V_5U波浅倒。7月3日上午心电图示:TV_1倒置较前变浅,TV_2呈正负双向,TV_(3-4)呈冠状T波,深达0.6～0.8mv,ST呈弓背向上,各导联U波明显变浅。7月4日上午心电图示:TV_2直立,TV_(3-4)呈正负双向(正向>负向),TV_5正常,各导联U波恢复正常,患者自觉症状明显好转,数天后出院。
Male patient, 59 years old. July 1, 1991 due to paroxysmal supranuclear area pain, with chest tightness for 3 days, post-event aggravating treatment. Had coronary heart disease history. Auscultation: low heart sound, no noise, law, blood pressure 14 / kPa, that is to do the ECG: T V_1 shallow down, TV_ (2-4) positive and negative bidirectional V_ (2-4) U deep wave down, up to 0.2 ~ 0.4mv, V_5U wave shallow down. On the morning of July 3, the electrocardiogram showed that TV_1 turned lighter than before, TV_2 showed positive and negative bidirectional, and TV_ (3-4) showed a coronary T wave with a depth of 0.6 ~ 0.8mv. Obviously lighter. On the morning of July 4, ECG showed: TV_2 was upright, TV_ (3-4) was positive and negative bidirectional (positive> negative), TV_5 was normal, U wave of each lead returned to normal, patients’ symptoms improved obviously and discharged several days later.