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各种肺外创伤常伴发肺损伤,休克过程更是如此,即使单纯失血性休克亦可因其迁延而死于肺衰。对此,多认为与感染后的内毒素作用有关。肠道是一个内毒素库,在休克过程有可能因屏障机能降低而导致肠道因素入侵;待其将肝网状内皮系统(RES)封闭后必将逃离肝脏而入肺造成损伤。已证明用印度墨水将家兔肝RES封闭和单纯失血性休克,短时内除均有白血细胞转入肝内屯积外,未见有明显肺损伤出现。但在预先人工封闭肝RES的基础上再造成急性失血性休
A variety of extra-pulmonary trauma often associated with lung injury, shock process is even more so, even simple hemorrhagic shock can also die due to its delay in pulmonary failure. In this regard, more thought to be related to the role of endotoxin after infection. The intestine is an endotoxin reservoir, which may lead to invasion of intestinal factors during the shock process due to the reduced barrier function. When the liver is closed, the liver reticular endothelial system (RES) will escape from the liver and cause damage to the lungs. Has proved that the Indian ink rabbit liver RES closed and hemorrhagic shock, in addition to short-term transfer of white blood cells into the liver, no obvious lung injury. However, in advance of artificial closure of liver RES on the basis of the resulting acute hemorrhagic Hugh