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(病例见本刊2011年第9期彩页)该患儿活化部分凝血活酶时间(APTT)时间延长,APTT主要反映内源性凝血是否正常,活化部分凝血活酶:(1)血浆因子Ⅷ,因子Ⅸ和因子XI水平减低,如血友病A、血友病B及因子XI缺乏症;(2)严重的凝血酶原(因子Ⅱ),因子Ⅴ、因子Ⅹ和纤维蛋白原缺乏,如阻塞性黄疽、肝脏疾病、新生儿出血症、肠道灭菌综合征、吸收不良综合征、口服抗凝剂及低(无)纤维蛋白血症等;(3)纤维蛋白溶解活力增强,如继发性、原发性纤维蛋白溶解功能亢进等;(4)
(See the case of this issue No. 9, 2011 9 color pages) of the activated partial thromboplastin time (APTT) in children with prolonged APTT mainly reflects the normal endogenous coagulation, activated partial thromboplastin: (1) plasma factor Ⅷ , Factor IX and factor XI levels, such as hemophilia A, hemophilia B and factor XI deficiency; (2) severe prothrombin (factor II), factor V, factor X and fibrinogen deficiency, such as Obstructive jaundice, liver disease, neonatal hemorrhagic disease, enteric sterility syndrome, malabsorption syndrome, oral anticoagulants and low (non) fibrinolytic disorders; (3) increased fibrinolytic activity such as Secondary, primary fibrinolysis hyperthyroidism, etc .; (4)