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本文对174例以重症肺结核为基础疾病的呼吸衰竭病例作回顾分析,结果表明:其中105例为Ⅰ型呼吸衰竭(占60.3％);67例失代偿性酸碱紊乱中,36例为呼碱型(占53.7％);53例死亡病例中,46例死于多脏器功能衰竭(占86.8％)。这显然有别于国内现有的以慢性阻塞性肺病为基础的呼吸衰竭资料。 1.重症肺结核导致呼衰的机制:(1)病变范围大。致使部分肺区末梢细支气管阻塞,闭陷,正常肺泡减少,而部分肺区毛细血管床面积减少,二者不成比例
In this paper, 174 cases of SARS-based respiratory failure were retrospectively analyzed. The results showed that 105 of them were type I respiratory failure (60.3%), and 67 of them were dysfunctional acid-base disorders (53.7%); of the 53 deaths, 46 died of multiple organ failure (86.8%). This is obviously different from the existing data on respiratory failure based on chronic obstructive pulmonary disease in China. 1. Severe tuberculosis lead to respiratory failure mechanism: (1) a wide range of lesions. Resulting in bronchial obstruction of the distal part of the lung, obstruction, decreased normal alveoli, and capillary area of some lung area decreased, both out of proportion