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急性心肌梗塞住院的心源性休克其发病率为10-15％。而普遍以心肌损害,少数为乳突肌断裂或穿孔所致。其中左室心肌梗塞占35-75％,其次是大面积心肌梗塞、主动脉逆流、冠状动脉侧枝通路的重建,或它的移植等。近10年的研究表明,早期治疗是预防心源性休克的重要关键。若发病3小时始治疗的其发病率为4％,而>3小时者则增加达13％。显然,减少心肌的损害即能降低休克发病率。若对梗塞程度不清楚,将对此休克的临床意义认识不足。据观测,凡有严重心瓣膜和左前降枝器质性损害者,或广泛心肌梗塞而致左室衰竭者,则有84％是濒死的心源性休克。心肌梗塞的发病机理经动物实验表明,冠脉狭窄的心肌梗塞其发病率颇高。若予手术后基本上能降低梗塞。临床实践也证明,心肌进行性损害必导致休克,同时也说明冠脉狭窄不仅导致心肌进行性
Acute myocardial infarction hospitalized cardiogenic shock its incidence rate of 10-15%. And generally to myocardial damage, a small number of mastoid muscle rupture or perforation. Left ventricular myocardial infarction accounted for 35-75%, followed by large myocardial infarction, aortic reflux, coronary collateral pathway reconstruction, or its transplantation. Nearly 10 years of research show that early treatment is an important key to prevent cardiogenic shock. The onset rate was 4% at 3 hours after onset and 13% at> 3 hours. Obviously, reducing myocardial damage that can reduce the incidence of shock. If the extent of infarction is not clear, the clinical significance of this shock lack of understanding. It is observed that where there are serious heart valves and left anterior descending organ damage, or extensive myocardial infarction caused by left ventricular failure, then 84% are dying cardiogenic shock. The pathogenesis of myocardial infarction Animal experiments show that the incidence of myocardial infarction coronary stenosis is high. If the surgery can basically reduce infarction. Clinical practice also shows that progressive myocardial damage will lead to shock, but also shows that coronary stenosis not only leads to myocardial progressive