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感染性休克的发病机制及治疗問題,虽經各家探索,迄未完滿解决。近年来多認为在发病初期,內毒素通过间接的作用,引致微循环(micro—Circulation)血管痙攣,血液滞留于內脏(特别是门靜脉系統)及皮肤,因而回心血量及心搏出量减少,导致休克。故在血管痙攣期可給血管舒张莉(如Dibezyline),以促使回心血量增加,糾正休克。錢潮氏首先报导靜脉注射大剂量阿托品可直接解除小血管痙
The pathogenesis and treatment of septic shock, although explored by each, has not yet been fully resolved. In recent years, many believe that in the early stages of onset, endotoxin indirectly causes micro-circulation (micro-circulation) vasospasm, blood retention in the visceral (especially the portal vein system) and the skin, so back to the amount of blood and stroke Reduce the amount, leading to shock. Therefore, vasoconstriction in the vasodilator Li (such as Dibezyline), to promote the return of blood volume, correct shock. Qian Chao first reported intravenous injection of large doses of atropine can be directly relieved of small vasospasm