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平滑肌细胞的增殖与游走视为经皮冠状动及成形术(PTCA)后再狭窄机制之一 [1,2 ]。既往研究着眼于机械或药物干预这一病理生理过程。但迄今为止尚未获得理想结果。随着分子生物学技术的发展 ,基因治疗 PTCA术后再狭窄遂引人注目 ,其相关研究报导已迭见文献 [1～ 8] 。本文采用腺
Proliferation and migration of smooth muscle cells are considered as one of the mechanisms of restenosis after percutaneous transluminal coronary angioplasty (PTCA) [1,2]. Previous studies have focused on the pathophysiological process of mechanical or drug intervention. But so far no satisfactory result has been achieved. With the development of molecular biology techniques, gene therapy of PTCA restenosis after attracting attention, the relevant research reports have been seen in the literature [1 ~ 8]. This article uses glands