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在三碘季胺酚制动下，对３９只清醒麻痹的家兔进行实验，探讨隔核对海马痛单位的作用及其可能机制。结果发现，海马的痛单位占引导总数的２８．３７％，其中以痛兴奋单位为主（占痛单位的７０．３４％）。海马有８０．５１％的痛单位对刺激隔核起反应，含痛兴奋性神经元（ＰＥＮ）７１个，痛抑制性神经元（ＰＩＮ）２４个。其中受隔核影响出现减频反应的海马痛单位有７２个，占７５．７９％，含ＰＥＮ６１个，ＰＩＮ１１个；受隔核影响出现增频反应的海马痛单位有２３个，占２４．２１％，含ＰＥＮ１０个，ＰＩＮ１３个。脑室注射阿托品或氟哌啶醇均可阻断刺激隔核对海马的影响效应。提示：隔核参与对海马痛单位的调制；刺激隔核对海马痛单位以抑制性调制为主，并主要作用于ＰＥＮ；乙酰胆碱、多巴胺及相应的受体可能参与其调制。
Thirty-three conscious paralyzed rabbits were experimented under the action of triiodide quaternary ammonium phenol to explore the effect of septal nuclei on the units of hippocampal pain and its possible mechanism. The results showed that the pain unit of the hippocampus accounted for 28.37% of the total number of the guide, of which the main unit of pain excitement (accounting for 70.34% of the pain unit). The hippocampus had 80.51% of the pain units responding to stimulating the septum. There were 71 PEN and 24 pain inhibitory neurons (PIN). Among them, there were 72 hippocampal pain units with frequency reduction reaction, accounting for 75.79%, including PEN61 and PIN11, and 23 units of hippocampal hyperactivity reaction with frequency of response to septal decoction, accounting for 24.21 %, Including PEN10, PIN13. Intraventricular injection of atropine or haloperidol can block the stimulation of septal effects on the hippocampus. Tip: septal involvement in the modulation of hippocampal pain units; stimulation of septal hippocampal pain units to inhibit the modulation of the main, and the main role in PEN; acetylcholine, dopamine and the corresponding receptors may participate in the modulation.