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作者报告1例66岁男性患者,1963年4月出现自发性皮肤瘀斑和气促,无化学物质接触史。体查:皮肤苍白和瘀斑,肝脾不大。实验室检查:全血细胞减少,网织红细胞<1%,血清B_(12)、叶酸浓度正常,骨髓增生低下,粒、红系明显受抑,巨核细胞缺如,分类以淋巴和浆细胞为主,血清结合珠蛋白正常,尿含铁血黄素阴性,诊断为再生障碍性贫血。用强的松60mg/日治疗,同年7月用睾丸酮治疗,均无明显疗效。因而先后3次输入胎儿肝细胞共50ml,并停用睾丸酮。此后病情逐渐好转,到1965年9月血象恢复正
The authors report a 66-year-old male patient with spontaneous skin ecchymosis and shortness of breath in April 1963 with no history of chemical exposure. Physical examination: pale skin and ecchymosis, hepatosplenism is not. Laboratory tests: pancytopenia, reticulocyte <1%, serum B_ (12), folic acid concentration was normal, low myeloid hyperplasia, granulosa, erythroid significantly inhibited, lack of megakaryocytes, lymphoid and plasma cell-based classification , Serum haptoglobin normal, negative hemosiderin, diagnosis of aplastic anemia. With prednisone 60mg / day treatment, in July the same year with testosterone treatment, no significant effect. Therefore, three times in a total input fetal liver cells 50ml, and disable testosterone. Since then the condition gradually improved, to September 1965 blood was recovered