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应用角叉菜胶后肢局部致炎作为炎症痛的实验动物模型，通过在细胞内记录技术，观察和分析了猫脊髓背根节Aβ神经元的自发放电特性和诱因。结果表明炎症痛导致猫背根节有自发放电的Aβ神经元数明显增加，并表现为持续高频、簇状和不规则等多种型式的放电；在炎症痛不同时程（4和12h），静息膜电位有一定程度的去极化；大多数有自发放电活动的神经元（91％）对外周机械性刺激表现为兴奋反应，其中有部分神经元（9％）同时具有兴奋性和抑制性感受野。胞体给予TTX能阻断这种自发放电；切断外周神经干能使这种自发放电消失，而切断其背根不影响这种自发放电活动。提示炎症痛猫背根节Aβ神经元的兴奋性增强与炎症部位持续性痛的存在相关，可能是炎症性触诱发痛（Allodynia）形成的基础。
Local inflammation of carrageenan hind limbs was used as an experimental animal model of inflammatory pain. The spontaneous firing characteristics and inducement of Aβ neurons in the dorsal root ganglia of the cat spinal cord were observed and analyzed by intracellular recording technique. The results showed that inflammatory pain led to a significant increase in the number of spontaneous Aβ neurons in the dorsal root ganglia and showed persistent high frequency, clustered and irregular discharges. At different time points (4 and 12 h) Resting membrane potential to a certain degree of depolarization; most spontaneous discharge activity of neurons (91%) on peripheral mechanical stimulation showed an excitatory response, in which some neurons (9%) both excitability and inhibition Sexy receptive field. TTX can block this spontaneous discharge; the removal of peripheral nerve stem can make this spontaneous discharge disappear, while cutting off the dorsal root does not affect this spontaneous discharge activity. It is suggested that the increased excitability of Aβ neurons in inflammatory pain cat dorsal root ganglion is related to the persistent pain in inflammatory sites and may be the basis for the formation of inflammatory allodynia.