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腹部外伤海水浸泡后急性炎症反应综合征的基本病理生理变化是机体内促炎与抗炎因子失衡所致的过度炎症反应。核因子κB(nuclear factor kappa B,NF-κB)是创伤后调节细胞炎症反应的许多基因的转录活化所必需的细胞因子。同时,作为NF-κB上游受体的Toll样受体4(TLR4),在炎症反应过程中能够激活以NF-κB为核心的
Abdominal trauma after seawater immersion acute inflammatory response syndrome, the basic pathophysiological changes in the body inflammation and anti-inflammatory factors imbalance caused by excessive inflammation. Nuclear factor kappa B (NF-κB) is a cytokine necessary for the transcriptional activation of many genes that regulate cellular inflammatory responses after trauma. In the meantime, Toll-like receptor 4 (TLR4), which is an upstream receptor of NF-κB, activates NF-κB as a core during the inflammatory reaction